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Vascular oxidative stress and nitric oxide depletion in HIV-1 transgenic rats are reversed by glutathione restoration.

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Low blood CD8+ T-lymphocytes and high circulating monocytes are predictors of HIV-1-associated progressive encephalopathy in children.

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Effect of heart failure on skeletal muscle myofibrillar protein content, isoform expression and calcium sensitivity.

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HIV-Tat protein induces oxidative and inflammatory pathways in brain endothelium.

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Primary T-cells from human CD4/CCR5-transgenic rats support all early steps of HIV-1 replication including integration, but display impaired viral gene expression.

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Effect of heart failure on the regulation of skeletal muscle protein synthesis, breakdown, and apoptosis.

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